YK11 Sarms powder CAS 431579-34-9

Raw Raw YK-11 powder (431579-34-9) Descripción

Many users think YK11 is the key to unlocking the body’s full potential and achieving fast muscle gains with few side effects. It also said that YK11 has the ability to reduce myostatin activity, making it a myostatin inhibitor. The YK-11 is definitely one of the strongest ones out there. You can’t really compare YK to LGD-4033 or any other products because of how strong it is.

Sin embargo, even though YK-11 raw powder is incredibly strong keep in mind that there have been only a few studies done with this compound. This is the main reason most people seem to rather go with a others such as Ostamuscle, RAD-140 or GW-501516.

YK11 is gaining interest from bodybuilders due to its ability to quickly build muscle with minimal side effects.

One cellular study suggests that YK11 increases muscle mass. Researchers found that it increases the muscle-building protein follistatin (a myostatin inhibitor).

YK11 may also enhance bone health. In a study on bone cells, YK11 increased the levels of bone-building proteins.

Some bodybuilders who have experimented with YK11 report great muscle gains and fat loss with minimal side effects. Sin embargo, there are currently no animal or human studies of YK11 and its safety is unclear.

Raw YK-11 powder (431579-34-9) Mechanism of Action

YK11 has a similar chemical structure to DHT and this is shown by the way it binds to androgen receptors. The body produces DHT naturally for the purpose of promoting prostate health, hair growth, and appropriate puberty development.

Researchers did a cell study to find out how it works to build muscle. The results show that it simply aims to increase follistatin levels. Follistatin is another naturally occurring protein in the human body which helps keep myostatin in check. Myostatin is a hormone whose job is to curtail unbridled muscle growth.

So far, only YK11 raw powder has the ability to inhibit myostatin.

Further evidence suggests that YK11 can also promote bone growth by interfering with a gateway that functions like DHT.